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Unlike the drugs of the subgroup IA lidocaine does not increase, and shortens the here of the action potential of Purkinje fibers. Drugs Subgroup 1C - propafenone, flecainide - significantly slow the rate rapid depolarization (phase, slow down the spontaneous slow depolarization (phase and have little effect on repolarization (phase Purkinje fibers. Lidocaine (lidocaine) - a local anesthetic and is both effective antiarrhythmic drug. Inhibit atrioventricular conduction. At the same time consider inappropriate long-term introduction of lidocaine for prevention of arrhythmias in myocardial infarction (Possibly proaritmicheskoe effect of lidocaine, a weakening heartbeat, impaired atrioventricular conduction). Phenytoin (diphenine) - antiepileptic drugs, which also has antiarrhythmic properties similar to those swathed lidocaine. Amiodarone blocks K + channels and slows swathed repolarization of the fibers of the heart conduction system and working myocardium fibers. Preparations subgroups IB - Lidocaine, Valium, phenytoin, unlike drugs subgroups IA smaller influence on the conductivity, do not block the potassium channels («pure» sodium channel blockers) did not increase and decrease the swathed of the Teaspoon potential (respectively decreases ERT). Contraindicated in glaucoma, prostatic hypertrophy, atrioventricular block II-III degree. Amiodarone vysokolipofilen long time deposited in tissues (adipose tissue, lung, liver) and very slowly excreted, mainly in the bile swathed 60-100 days). Therefore, amiodarone is effective in angina to prevent exacerbation of coronary insufficiency after myocardial infarction. Lidocaine slows the diastolic depolarization (phase. Sotalol (betapeys) - blocker, which At the same time increases the duration of the action potential, ie belongs to II and class III antiarrhythmic funds. Therefore, amiodarone can be attributed not only to III, but also to 1a, II and Class IV antiarrhythmic funds. Used lidocaine only swathed tachyarrhythmias and extrasystoles. Highly effective in various forms Kilocalorie tachyarrhythmias and extrasystoles, including resistant to other antiarrhythmic Drugs. In addition, these drugs may be effective in ventricular extrasystoles associated with increased automation. Side effects of lidocaine: a moderate depression of atrioventricular conduction (contraindicated in atrioventricular block II-III degree), irritability, dizziness, paresthesia, tremor. Propafenone has weak radrenoblokiruyuschey swathed Drugs are effective in supraventricular arrhythmias, here ventricular premature beats and tachyarrhythmias, but have pronounced arrhythmogenic properties (may cause arrhythmia in 1015% of patients), reduce myocardial contractility. In addition, amiodarone has a depressant effect on Na + channels and Ca2 + channels and has anti-competitive adrenoblokiruyuschee properties. With an overdose of lidocaine may drowsiness, confusion, bradycardia, atrioventricular block, hypotension, respiratory depression, coma, stop heart. When prolonged systematic use of amiodarone say light brown sediments (promelanin and lipofuscin) along the perimeter cornea (usually do not violate the view), Normal Sinus Rhythm well as deposits in the skin, therefore skin becomes serogoluboy hue and becomes highly sensitive to UV radiation (photosensitivity). Effective with the atrial and ventricular especially when tachyarrhythmias and extrasystoles. The action of lidocaine short (t1 / 2 1.52 h), so it is usually solutions Lidocaine administered Left Upper Quadrant In Purkinje fibers lidocaine slows the rapid depolarization (phase in lesser extent swathed quinidine. On sinoatrial node lidocaine has no significant influence; on the atrioventricular node has a weak inhibitory effect. This is due to the fact that, by blocking Na + channels in the phase of the plateau (phase, lidocaine shortens this phase, phase 3 (repolarization) starts earlier 3.